SEGH Articles

Arsenic Biogeochemistry and Health

04 November 2014
The success of the 29th SEGH conference produced a special issue of papers presenting recent advances in various aspects of environmental and health impacts of contaminants, published in Environmental Geochemistry and Health

 

The 29th international Conference for Environmental Geochemistry and Health was held in Toulouse, France, from July 8th to 12th 2013 (http://segh2013.sciencesconf.org/). This annual meeting of the SEGH brought together 160 scientists from 35 countries to exchange ideas and results about regular topics (Biogeochemistry and Health, Biogeochemistry and Ecotoxicology, Spatial and Temporal Records of Pollution including Catchment Studies), as well as in Special Sessions (Frontiers in Mercury Biogeochemistry, Bioaccessibility of Pollutants in Soils and Vegetables, Arsenic: Current Issues of Speciation, Environmental Behaviour and Human Health Impacts). The participants had the occasion to present their work as well as learn from colleagues during three days of parallel sessions and outdoor poster sessions under the warm sun of Toulouse.


The success of this 29th event had several outcomes in the form of special issues. The first special issue was a collection of thirteen original papers presenting recent advances in various aspects of environmental and health impacts of contaminants, published in Environmental Geochemistry and Health, the SEGH Journal (http://link.springer.com/journal/10653/36/5/page/1). All the SEGH members have free access to this journal. A second special issue, published in Environmental Chemistry, grouped papers presented in one of the three special sessions, which was dealing with arsenic biogeochemistry and health, as well as the state of the art works in arsenic research (http://www.publish.csiro.au/nid/188/OnlineEarlyFlag/1.htm).

We encourage the SEGH members to take a closer look at these two special issues as well as to participate in future SEGH events and submit papers to our journals!

On behalf of the 29th SEGH conference organising committee as well as the guest editors of these two special issues, we warmly thank all the contributors for their valuable inputs.


Francois De Vleeschouwer & Kevin Francesconi. 

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Latest on-line papers from the SEGH journal: Environmental Geochemistry and Health

  • Genotoxic effects of PM 10 and PM 2.5 bound metals: metal bioaccessibility, free radical generation, and role of iron 2018-10-09

    Abstract

    The present study was undertaken to examine the possible genotoxicity of ambient particulate matter (PM10 and PM2.5) in Pune city. In both size fractions of PM, Fe was found to be the dominant metal by concentration, contributing 22% and 30% to the total mass of metals in PM10 and PM2.5, respectively. The speciation of soluble Fe in PM10 and PM2.5 was investigated. The average fraction of Fe3+ and Fe2+ concentrations in PM2.5 was 80.6% and 19.3%, respectively, while in PM2.5 this fraction was 71.1% and 29.9%, respectively. The dominance of Fe(III) state in both PM fractions facilitates the generation of hydroxyl radicals (·OH), which can damage deoxyribose nucleic acid (DNA), as was evident from the gel electrophoresis study. The DNA damage by ·OH was supported through the in silico density functional theory (DFT) method. DFT results showed that C8 site of guanine (G)/adenine (A) and C6 site of thymine (T)/cytosine (C) would be energetically more favorable for the attack of hydroxyl radicals, when compared with the C4 and C5 sites. The non-standard Watson–Crick base pairing models of oxidative products of G, A, T and C yield lower-energy conformations than canonical dA:dT and dG:dC base pairing. This study may pave the way to understand the structural consequences of base-mediated oxidative lesions in DNA and its role in human diseases.

  • A systematic review on global pollution status of particulate matter-associated potential toxic elements and health perspectives in urban environment 2018-10-08

    Abstract

    Airborne particulate matter (PM) that is a heterogeneous mixture of particles with a variety of chemical components and physical features acts as a potential risk to human health. The ability to pose health risk depends upon the size, concentration and chemical composition of the suspended particles. Potential toxic elements (PTEs) associated with PM have multiple sources of origin, and each source has the ability to generate multiple particulate PTEs. In urban areas, automobile, industrial emissions, construction and demolition activities are the major anthropogenic sources of pollution. Fine particles associated with PTEs have the ability to penetrate deep into respiratory system resulting in an increasing range of adverse health effects, at ever-lower concentrations. In-depth investigation of PTEs content and mode of occurrence in PM is important from both environmental and pathological point of view. Considering this air pollution risk, several studies had addressed the issues related to these pollutants in road and street dust, indicating high pollution level than the air quality guidelines. Observed from the literature, particulate PTEs pollution can lead to respiratory symptoms, cardiovascular problems, lungs cancer, reduced lungs function, asthma and severe case mortality. Due to the important role of PM and associated PTEs, detailed knowledge of their impacts on human health is of key importance.

  • Interactions between polycyclic aromatic hydrocarbons and epoxide hydrolase 1 play roles in asthma 2018-10-06

    Abstract

    Asthma, as one of the most common chronic diseases in children and adults, is a consequence of complex gene–environment interactions. Polycyclic aromatic hydrocarbons (PAHs), as a group of widespread environmental organic pollutants, are involved in the development, triggering and pathologic changes of asthma. Various previous studies reported the critical roles of PAHs in immune changes, oxidative stress and environment–gene interactions of asthma. EPHX1 (the gene of epoxide hydrolase 1, an enzyme mediating human PAH metabolism) had a possible association with asthma by influencing PAH metabolism. This review summarized that (1) the roles of PAHs in asthma—work as risk factors; (2) the possible mechanisms involved in PAH-related asthma—through immunologic and oxidative stress changes; (3) the interactions between PAHs and EPHX1 involved in asthma—enzymatic activity of epoxide hydrolase 1, which affected by EPHX1 genotypes/SNPs/diplotypes, could influence human PAH metabolism and people’s vulnerability to PAH exposure. This review provided a better understanding of the above interactions and underlying mechanisms for asthma which help to raise public’s concern on PAH control and develop strategies for individual asthma primary prevention.